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Qucik Contact
+91 805-166-2233
Monday to Sat Day
09.00 to 17.00
In this video Dr Rajeev Sharan introduces the common causes of oral cancer. That a majority of oral cancer cases in India are men and 90% of these cases are caused due to consumption of tobacco. He also mentions that's this consumption may be in the form of smoking or non-smoking. Consumption of Pan Masala, Khaini, Gutka, etc. may lead to oral cancer. Repeated injuries caused to the mucosa lining of the mouth causes oral cancer. White or red patches in the mouth that lead to difficulty opening mouth are precancerous symptoms. If persistent ulcers in the mouth that refuse to heal in 2 weeks are found, it could be a sign of oral cancer. Biopsies might confirm cancers. Early detection may lead to a cure and quitting tobacco along with treatment has been known to reverse the effects to pre-cancer stage. Avoiding tobacco for 10 years has been known to heal the body to the extent of being a person who has never taken tobacco.
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In this video Dr Sharan talks about oral cancer. He begins by elaborating the anatomy of oral cavity and naming the common causes behind oral cancer such as smoking, khaini, gutka, pan masala, etc. Elaborate the role of tobacco in carcinogenesis (3:29). Dr Sharan then explains the ways to reverse the effects of tobacco. The potentially malignant lesions due to continued use of tobacco are leukoplakia, erythroplakia, palatal lesion in reverse smoker and submucous fibrosis. The most important methods to manage oral cancer are biopsy, removal of predisposing habits (smoking, consuming tobacco), topical and systemic treatment, surgical treatment, and regular follow-up. Dr Sharan presents data which shows that 556, 400 cancer deaths happened in India in a particular year. 40% of cancers in India are oral cancer, 90% of which is caused by chewing tobacco. Symptoms are non-healing ulcer in the mouth, white or red patches in mouth, loose teeth, swelling in neck, bleeding from mouth, difficulty in chewing and swallowing. Diagnosis of Cancer has often been known to lead to troublesome emotional, psychological, financial and social impact for the patient. Dr Sharan specifies that biopsy is essential and does not spread the cancer. Oral cancer can be cured and the results depend upon the stage at which it is detected. Message to investigate our biopsy, imaging (CT/MRI) and metastatic work up (CxR, CT chest, PET-CT). Multidisciplinary approach is recommended to cure cancer. Surgery is the main treatment for cancer. For early lesions only surgery is required. For advanced lesions, surgery is accompanied with adjuvant Radiotherapy or Chemo-Radiotherapy. Prognostic markers are pathological involvement of cervical nodes, extracapsular spread, T stage, tumour thickness, surgical margins, pattern of tumour invasion and presence of perineural lymphovascular invasion. The survival rate in India has been known to be 20% less than that in the US. Dr Sharan in his presentation by presenting methods to self-examine the oral cavity for signs of cancer.
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In this video Dr Sharan introduces the signs and symptoms of head and neck cancer. People who smoke or consume tobacco are more likely to get head and neck cancer than those who do not. Consumptions pan masala, gutka, cigarettes, biris, zarda, khaini, etc. are common causes. The easy availability of tobacco in India due to us being the third largest producer and consumer of tobacco in the world, it has been found that 35% of the total adult population in India used tobacco and 14.1% of children between the ages of 13 to 15 use tobacco (mainly smokeless in the latter case). Dr Sharan presence data that shows the 71% of cancer deaths in India happen in people aged between 30 and 69 years. The major causes were oral (22.9%), stomach (12.6$) and lung (11.4%) in men, and cervical (17.1%), stomach (14.1%) and breast (10.2%) in women. The data shows that there were twice as many deaths from oral cancer than lung cancer. Dr Sharan then goes on to elaborate the process of tobacco causing cancer in people (4:40). The potentially malignant lesions are leukoplakia, erythroplakia, palatial lesion in reverse smoker, submucous fibrosis. The treatment of lesions are an immediate stop in tobacco consumption and consulting an oncologist. The symptoms are white or red patches in mouth, non-healing ulcer in mouth, swelling in neck, bleeding from mouth or nose, nasal blocks, difficulty in swallowing, hoarseness of voice and difficulty in breathing. Dr Sharan then presents a case study of a 52-year-old male teacher. Dr Sharan busts a few myths. Biopsy is essential and it does not aid the growth of cancer. Oral cancer can be cured and results depend on the stage of cancer. Dr Sharan adds that several public education campaigns are helping people recognise the early signs of the disease and are encouraging to seek medical attention for better prognosis. Such early detection of Cancer can save lives. More than a third of all cancers are preventable by reducing exposure to risk factors that include tobacco, obesity, physical inactivity, and sexually transmitted diseases. Screening programmes are available to detect cancer early.
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In this video Dr Rajeev Sharan discusses the goals of initial therapy of differentiated thyroid cancer (DTC). He begins by mentioning that the first goal is to remove the primary tumour and the nodal mets, the second being minimising the risk of recurrence and metastatic spread of the disease. Facilitating Radioactive Iodine Ablations (RAI) where appropriate, permitting accurate staging and risk stratification of the disease, and minimising treatment-related morbidity are the other goals of DTC. SEER Data can show that in 5 years, the relative survival rate of patients was over 98%. He mentions that in 2005, certain changes in the ATA guidelines were made for. They now recommend less aggressive surgery for low-risk thyroid cancer Central compartment node excision optional for low-risk thyroid cancer. The guidelines also mention that less than 1 cm cancer, if unifocal, without a radiation history or syndromal PTC only needs lobectomy and no lymph node sampling. Active surveillance could be considered in certain cases. Dr Sharan then elaborates on total thyroidectomy (2:19) and presents a case study on a 55-year-old lady with a 2-year-old hemoptysis. Dr Sharan then introduces the practice of scarless thyroidectomy. Robotic or endoscopic thyroidectomy include the breast axillary approach with CO2 gas, gasless axillary approach and retro-auricular approach which give better cosmetic satisfaction and have been acknowledged as safe as open thyroidectomy. Dr Sharan conclude by saying that the choice of surgical procedure can be tailored to the oncological needs of the patient. Surgical morbidity should be minimal. Remote access robotic or endoscopic thyroidectomy is safe and the ontological outcome is great with better cosmesis.
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